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	题名	作者	年代	出处	被引量
1	干扰素调节因子5通过损伤胞葬作用控制动脉粥样硬化病变中的坏死核心形成显示文摘髓系细胞是动脉粥样硬化病变发展和易损斑块形成的核心。动脉吞噬细胞吸收凋亡细胞(胞葬作用)的能力受损,可促进病变生长并建立坏死核心。干扰素调节因子(interferon regulatory factor,IRF)5是髓系细胞功能的重要调节因子。该研究试图确定IRF5是否影响动脉粥样硬化病变的形成和表型。方法:通过两种互补模型,研究IRF5在动脉粥样硬化中的作用:	Seneviratne AN Edsfeldt AO Cole JE Kassiteridi C Swart M Park I Green P Khoyratty TE Saliba DG Goddard ME Sansom SN Goncalves I Krams R Udalova IA Monaco C 刘莉叶鹏	2017	中华高血压杂志2017，25，8：	9
2	Interaction of AP-1 with a cluster of NF-kappa B binding elements in the human TNF promoter re- gion显示文摘	Udalova I A Kwlatkowski D	2001	Biochem Biophys Res Commun2001，289，1：	1
3	Similarities and differences between human and murine TNF promoters in their response to lipopolysaccharide 显示文摘	Udalova IA Turetskaya RL	1999	J Immunol1999，162，：	1
4	Evidence for a dual mechanism for IL-10 suppression of TNF-alpha production that does not involve inhibition of p38 mitogen-activated protein kinase or NF-kappa B in primary human macrophages显示文摘	Denys A Udalova IA Smith C	2002	J Immunol2002，168，10：	1
5	A polymorphism that affects OCT-1 binding to the TNF promoter region is associated with severe malaria 显示文摘	KNIGHT J C UDALOVA I HILL A V	1999	Nat Genet1999，22，2：	1
6	Functional consequences of a polymorphism affecting NF-kappaB p50-p50 binding to the TNF promoter region 显示文摘	UDALOVA I A RICHARDSON A DENYS A	2000	Mol Cell Biol2000，20，24：	1
7	Functional consequences of a polymorphism affecting NF-kappaB p50-p50 binding to the TNF promoter region 显示文摘	Udalova IA Richardson A Denys A	2000	Mol Cell Biol2000，20，24：	1
8	A polymorphism that affects OCT-1 binding to the TNF promoter region is associated with severe malaria显示文摘	Udalova I Hill AV	1999	Nat Genet1999，22，2：	1
9	Functional consequences of a polymorphism affecting NF-κB p50-p50 binding to the TNF promoter region显示文摘	Udalova IA Richardson A Denys A	2000	Mol Cell Bio12000，20，24：	1
10	A polymorphism that affects OCT-1 binding to the TNF promoter region is associated with severe malaria显示文摘	Knight JC Udalova I Hill AV		0，，02：	1
11	-308 tumor necrosis factor (TNF) polymorphism is not associated with survival in severe sepsis and is unrelated to lip polysaccharide inducibility of the human TNF promoter显示文摘	Udalova IA Book M	1996	J Inflamm1996，46，1：	1
12	Inflammatory bowel disease is associated with a TNF polymorphism that affects an interaction between the OCT1 and NF-κB transcription factors 显示文摘	Van Heel DA Udalova IA De Silva AP	2002	Hum Mol Genet2002，11，：	1
13	Functional consequences of a polymorphism affecting NF-kappaB-p50 binding to the TNF promoter region显示文摘	Udalova IA Richardson A Denys A	2000	Mol Cell Biol2000，20，24：	1
14	Quantitative prediction of NF-kappa B DNA-protein interactions显示文摘	Mott R Field D	2002	Proc Natl Acad Sci USA2002，99，：	1
15	Functional implications of genetic variation in no coding DNA for disease susceptibility and gene regulation显示文摘	Knight JC Louis E Udalova I	2003	Clin Sci2003，104，5：	1
16	Inflammatory bowel disease is associated with a TNF polymorphism that affects an interaction between the OCT1 and NF-KB transcription factors显示文摘	van Heel DA Udalova IA De Silva AP	2002	Hum Mol Genet2002，11，11：	1
17	Highly informative typing of the human TNF locus using siz adjacent polymorphic markes显示文摘	Udalova IA Nedospasov SA Webb GC	1993	Genomics1993，16，：	1
18	显示文摘	Udalova O V Tulenin Y P	2000	Catal Lett2000，69，：	1
19	Functional consequences of a polymorphism affecting NF-kappa B p50-p50 binding to the TNF promoter region显示文摘	Udalova IA Richardson A Denys A	2000	Mol Cell Biol2000，20，：	1
20	-308 tumor necrosis factor (TNF) polymorphism is not associated with survival in severe sepsis and is unrelated to lipopolysaccharide inducibility of the human TNF promoter显示文摘	Stuber F Udalova IA Book M	1996	J Inflamm1996，46，1：	1

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