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6篇 您的检索式:作者名="Torroja L"
    题名 作者 年代 出处 被引量
1The Drosophila β-amyloid precursor protein homolog promotes synapse differentiation at the neuromucscular junction 显示文摘Torroja L Packard M Gorczyca M White K Budnik V 1999J Neurosci1999,19,:1
2Loss of glial lazarillo,a homolog of apolipoprotein D,reduces lifespan and stress resistance in Drosophila显示文摘Sanchez D Lopez-Arias B Torroja L 2006Curr Biol2006,16,7:1
3scully, an essential gene of Drosophila,is homologous to mammalian mitochondrial type II L-3-hydroxyacyl-CoA dehydrogenase/amyloid-beta peptide-bind- ing protein 显示文摘Torroja L Ortufio-Sahagfn D Ferr~s A 1998J Cell Bio11998,141,4:1
4Neuronal overexpression of APPL, the drosophila homologue of the amyloid precursor protein (APP), disrupts axonal transport 显示文摘Torroja L Chu H Kotovsky I 1999Curr Biol1999,9,:1
5Abnormal muscle development in the heldup3 mutant of Drosophila melanogaster is caused by a splicing defect affecting selected troponin I isoforms显示文摘BARBAS J A GALCERAN J TORROJA L 1993Molecular and Cellular Biology1993,13,3:1
6The Drosophila adult neuromuscular junction as a model for unravelling amyloid peptide influence on synapse dynamics显示文摘Amyloid peptide(Aβ)oligomers are considered one of the primary causal factors for the synaptic loss characteristic of Alzheimer’s disease(AD)(Karran and De Strooper,2016).However,Aβis generated in normal brains and accumulates at synaptic sites,which raises the question whether Aβplays a physiological role in synapses.This unresolved issue is especially relevant in viewBegona López-Arias Ignacio Monedero Enrique Turiégano Laura Torroja 2017Neural Regeneration Research2017,12,12:0
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