|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | 显示文摘 | Lafuente MP Villegas-Perez MP Sobrado-Calvo P : Neuroprotective effects of alpha (2)-selective adrenergic agonists against ischemiainduced retinal ganglion cell death | 2001 | Invest Ophthalmol Vis Sci2001,42,: | 1 |
| 2 | Pneumatic retinopexy:a multicenter randomized controlled clinical trial comparing pneumatic retinopexy with scleral buckling显示文摘 | Tornambe PE Hilton GF the Retinal Detachment StudyGroup | | 0,,06: | 1 |
| 3 | Natural history and clinical management of central retinal vein occlusion:The central vein occlusion study显示文摘 | | 1997 | Arch Ophthalmol1997,115,: | 1 |
| 4 | A theory based on oxygen and vascular physiology 显示文摘 | Stefansson E The therapeutic effects of retinal laser treatment and vitrectomy | 2001 | Acta Ophthahnl Scand2001,79,: | 1 |
| 5 | Beneficial injury in obese type 2 diabeticrats显示文摘 | MAEDA S YAMAGISHI S effects of vildagliptin on retinal MATSUI T | 2013 | Ophthalmic Res2013,50,4: | 1 |
| 6 | Neuropr0tec- tion in Glaucoma显示文摘 | Glaucoma Retinal Degeneration Research Group | 2008 | Optom Vis Sci2008,85,6: | 1 |
| 7 | The classification of retinal detachment with proliferative vitreoretinopathy显示文摘 | The Retinal society terminology Committee | 1983 | Ophthalmology1983,90,: | 1 |
| 8 | Intraocular pressure during transient exposure to microgravity显示文摘 | Mader TH Gibson CR Caputo and retinal vascular changes M | 1993 | Am J Ophthalmol1993,115,3: | 1 |
| 9 | The eye disease case-control study group显示文摘 | Risk factors for branch retinal vein occlusion | 1993 | Am J Ophthalmol1993,116,3: | 1 |
| 10 | Clinical variables associated with failure of retinal de- tachment repair:the European vitreo-retinal society retinal de- tachment study report number 4 显示文摘 | Adelman RA Parnes A J Michalewska Z Ducoumau D Europe- an Vitreo-Retinal Society (EVRS) Retinal Detachment Stud Group | 2014 | Ophthalmology2014,121,9: | 1 |
| 11 | The classification of retinal detachment with proliferative vitreoretinopathy 显示文摘 | The Retinal Society Terminolgy Committee | 1983 | Ophthalmology1983,90,2: | 1 |
| 12 | The role and timing of pars plana vitrectomy in penetrating ocular trauma 显示文摘 | Proliferative vitreoretinopathy : Process in Retinal and Eye Mieler WF Mittra RA | 1997 | Arch Ophthalmol1997,115,: | 1 |
| 13 | Evolution of dipolarization in the near-Earth current sheet induced by Earthward rapid flux transport显示文摘 | Nakamura R. Retinò A. Baumjohann W. Volwerk M. Erkaev N. Klecker B. Lucek E. A. Dandouras I. André M. Khotyaintsev Y | 2009 | Annales Geophysicae2009,,221: | 1 |
| 14 | w- derived mesenehymal stem ceils 显示文摘 | Chung JK Park TK Ohn YH et aL Modulation of retinal wound healing by systemically administered bone rear | 2011 | Korean J Ophthal- tool2011,25,4: | 1 |
| 15 | A randomized trial comparing the efficacy and safety of intravitreal triamcinolone with observation to treat vision loss associated with macular edema secondary to central retinal vein occlusion显示文摘 | The Standard Care vs Corticosteroid for Retinal Vein Occlusion (SCORE) Study Report 5 | 2009 | Arch Ophthalrnol2009,127,9: | 1 |
| 16 | Retinal survival time显示文摘 | Hayreh SS Zimmerman MB Kimura A Central retinal artery occlusion | 2004 | Exp Eye Res2004,78,: | 1 |
| 17 | Suofu Qin’s work on studies of cell survival signaling in cancer and epithelial cells显示文摘Reactive oxygen species (ROS) encompass a variety of diverse chemical species including superoxide anions, hydrogen peroxide, hydroxyl radicals and peroxynitrite, which are mainly produced via mitochondrial oxidative metabolism, enzymatic reactions, and light-initiated lipid peroxidation. Over-production of ROS and/or decrease in the antioxidant capacity cause cells to undergo oxi- dative stress that damages cellular macromolecules such as proteins, lipids, and DNA. Oxidative stress is associated with ageing and the development of agerelated diseases such as cancer and age-related macular degeneration. ROS activate signaling pathways that promote cell survival or lead to cell death, depending on the source and site of ROS production, the specific ROS generated, the concentration and kinetics of ROS generation, and the cell types being challenged. However, how the nature and compartmentalization of ROS contribute to the pathogenesis of individual diseases is poorly understood. Consequently, it is crucial to gain a comprehensive understanding of the molecular bases of cell oxidative stress signaling, which will then provide novel therapeutic opportunities to interfere with disease progression via targeting specific signaling pathways.Currently, Dr. Qin's work is focused on inflammatory and oxidative stress responses using the retinal pigment epithelial (RPE) cells as a model. The study of RPE cell inflammatory and oxidative stress responses has successfully led to a better understanding of RPE cell biology and identification of potential therapeutic targets. | Suofu Qin, Retinal Disease Research, Department of Biological Sciences, Allergan, Inc., 2525 Dupont Drive, Irvine, CA 92612-1599, United States | 2010 | World Journal of Biological Chemistry2010,1,12: | 1 |
| 18 | The Eye Disease Case-Control Study显示文摘 | Risk factors for central retinal vein occlusion | 1996 | Group Arch Ophthalmol1996,114,: | 1 |
| 19 | The classification of retinal detachment with proliferative vitreoretinopathy显示文摘 | The Retinal Society Terminology Committee | 1983 | Ophthalmology1983,90,: | 1 |
| 20 | Incidence and cpidemiological characteristics of rhegmatogenous retinal detachment in Beijing, China 显示文摘 | Li X Beijing Rhegmatogenous Retinal Detachment Study Group | 2003 | Ophthahnology2003,110,12: | 1 |