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52篇 您的检索式:作者名="RAVINGEROVA T"
    题名 作者 年代 出处 被引量
1The role of PPAR in myocardial response to ischemia in normal and diseased heart显示文摘Ravingerova T Adameova A Carnicka S 2011Gen Physiol Biophys2011,30,4:1
2The role of proteinkinases in responses to chronic social stress in rat hearts显示文摘Barancik M ivanova M ravingerova T 2009Physiol Res2009,68,:1
3Oxytocin exerts protective effects on in vitro myocardial injury induced by ischemia and reperfusion 显示文摘ONDREJCAKOVA M RAVINGEROVA T BAKOS J 2009Canadian Journal of Physiology and Pharmacology2009,87,2:1
4Oxytocin exerts protective effects on in vitro myocardial injury induced by ischemia and reperfusion显示文摘Ondrejcakova M Ravingerova T Bakos J 2009Can J Physiol Pharmacol2009,87,2:1
5Brief, intermediate and prolonged ischemia in the isolated crystalloid perfused rat heart: relation between susceptibility to arrhythmias and degree of ultrastructural injury显示文摘Ravingerova T Narcis T Slezak J 1995J Mol Cell Cardiol1995,27,9:1
6Preconditioning modulates susceptibility to ischemia-indueed arrhythmias in the rat heart: the role of al-adrenergic stimulation and KAT1, channels 显示文摘Ravingerova T Pancza D Ziegelhoffer A 2002Physiol Res2002,51,:1
7Brief,intermediate and prolonged ischemia in the isolated crystalloid perfusod rat heart:relationship between susceptibility to arrhythmias and degre of ultrastruetural injury显示文摘Ravingerova T Tribulova N Slezak J 1995J Mol Cell Cardiol1995,27,9:1
8Brief, intermediate and Ira)longed ischemia in the isolated crystalloid perfused rat heart: rela- tionship between susceptibility to arrhythmias and degree of ultrastrue- rural injury 显示文摘Ravingerova T Tribulova N Slezak J 1995J Mol Cell Cardiut1995,27,9:1
9Free oxygen radicals contribute to high incidence of reperfusion induced arrhythmias in isolated rat heart 显示文摘Ravingerova T Slezak J Tribulova N 1999Life Sci1999,65,1819:1
10The role of PPAR in myocardial response to ischemia in normal and diseased heart 显示文摘Ravingerova T Adameova A Camicka S 2011Gen Physiol Bio- phys2011,30,4:1
11Brief, intermediate and prolonged ischemia in the isolated crystalloid perfused rat heart: relationship between susceptibility to arrhythmias and degree of ultrastructural injury显示文摘Ravingerova T Tribulova N Slezak J 1995J Mol Cell Cardiol1995,27,9:1
12Brief, intermediate and prolonged isehemia in the isolated crystalloid perfused rat heart: relationship between susceptibility to arrhythmias and degree of uhrastruetural injury显示文摘Ravingerova T Tribulova N Slezak J 1995J Mol Cell Cardiol1995,27,9:1
13Free oxygen radicals contribute to high ineidence of repedusion-induced arrhythmias in isolated rat显示文摘 1999Heart Life Sci1999,65,1819:1
14Brief, intermediate and prolonged ischemia in the isolated crystalloid perfused rat heart: relationship between susceptibility to arrhythmias and degree of ultrastructurat injury 显示文摘RAVINGEROVA T TRIBULOVA N SLEZAK J et at 1995J Mol Cell Cardiol1995,27,9:1
15Brief,intermediate and prolonged ischemia in the isolated crystalloid perfused rat heart:relationship between susceptibility to arrhythmias and degree of ultrastructural injury显示文摘Ravingerova T Tribulova N Slezak J 1995J Mol Cell Cardiol1995,27,9:1
16Brief,intermediate and prolonged ischemia in the isolated crystalloid perfused rat heart :relationship between susceptibility to arrhythmias and degree of ultrastructural injury显示文摘Ravingerova T Tribulova N Slezak J 1995J Mol Cell Cardiol1995,27,9:1
17Partial prevention of calcium paradox in isolated pedused rat hearts by diltiazem显示文摘 Ravingerova T Tribulova N 1989Biomed Biochim Acta1989,48,23:1
18Mitogen-activated protein kinases:a new therapeutic target in cardiac pathology显示文摘RAVINGEROVA T BARANCIK M STRNISKOVA M 2003Mol Cell Biochem2003,247,12:1
19Intrinsic defensive mechanisms in the heart:a po- tential novel approach to cardiac protection against ischemic inju- ry 显示文摘Ravingerova T 2007Gen Physiol Biophys2007,26,1:1
20Intrinsic defensive mechanisms in the heart: a potential novel approach to cardiac protection against ischemic injury 显示文摘Ravingerova T 2007Gen Physiol Biophys2007,26,1:1
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