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| 1 | Management of massive hemoptysis显示文摘 | Wedzicha JA Pearson MC | 1990 | Respir Med1990，84，： | 1 |
| 2 | A comparison of the adhesion of mammalian cells and staphylococcus epidermidis on fibronectin-modified polymer surfaces显示文摘 | Dexter SJ Pearson RG Davies MC | 2001 | J Biomed Mater Res2001，56，2： | 1 |
| 3 | Spontaneous Cerebrospinal fluid rhinorrhea: Evolving concepts in diagnosis and surgical management based on the Mayo Clinic Experience from 1970 through 1981显示文摘 | Mc Donald TJ Pearson BW | 1985 | Neurosurgery1985，16，3： | 1 |
| 4 | Vasoactive mediator release by fetal endothelial cells in intrauterine growth restriction and preeclampsia显示文摘 | Parra MC Lees C Mann GE Pearson JD Nicolaides KH | 2001 | Am J Obstet Gynecol2001，184，3： | 1 |
| 5 | Treatment of eorticosteroid- resistant polymyositis with methotrexate显示文摘 | Sokoloff MC Goldberg LS Pearson CM | 1971 | Lancet1971，1，： | 1 |
| 6 | Epoxy / polyimide adhesion studies显示文摘 | Pearson R A Hoontrakul P Mc Adams B J | 2004 | PMSE Preprints2004，90，： | 1 |
| 7 | 大咯血的处理显示文摘大咯血是死亡率很高的急症,如果出血区域局限,病人具有手术指征,应选择手术治疗.不宜手术的病人可用支气管动脉栓塞疗法,以控制威胁生命的大咯血. | Wedzicha JA Pearson MC 朱同刚 程晔 | 1992 | 国外医学(内科学分册)1992，19，3： | 1 |
| 8 | Strangulated diaphragmatic hernia complicating delivery显示文摘 | Pearson SC Pillsbure SG Mc callum M | 1950 | J Am Med Assoc1950，144，1： | 1 |
| 9 | Treatment of corticosteroid-resistant polymyositis with methotrexate 显示文摘 | Sokoloff MC Goldberg LS and Pearson CM | 1971 | Lancet1971，1，7688： | 1 |
| 10 | Hypo-activity induced skeletal muscle atrophy and potential nutritional interventions: A review显示文摘Periods of hypo-activity result in profound changes in skeletal muscle morphology and strength. This review primarily addresses the differential impact of de-training, bed-rest, limb immobilisation and unilateral lower limb suspension on muscle morphology, strength and fatigability. The degree of muscle atrophy differs depending on the hypo-activity model and the muscles in question, with the leg and postural muscles being the most susceptible to atrophy. Hypo-activity also results in the dramatic loss of strength that often surpasses the loss of muscle mass, and consequently, the nervous system and contractile properties adapt to adjust for this excessive loss of strength. In addition, the degree of muscle strength loss is different depending on the hypo-activity model, with immobilisation appearing to have a greater impact on strength than unloaded models. There is a step-wise difference in the magnitude of muscle loss so that, even after accounting for differential durations of interventions immobilisation ≥ unilateral lower limb suspension ≥ bed-rest ≥ de-training. Muscle fatigability varies between hypoactivity models but the results are equivocal and thismay be due to task-specific adaptations. This review also addresses potential nutritional interventions for attenuating hypo-activity induced muscle atrophy and strength declines, in the absence of exercise. Essential amino acid supplementation stands as a strong candidate but other supplements are good contenders for attenuating hypo-activity induced atrophy and strength losses. Several potential nutritional supplements are highlighted that could be used to combat muscle atrophy but extensive research is needed to determine the most effective. | Emma L Bostock Christopher I Morse Keith Winwood Islay Mc Ewan Gladys L Onambélé-Pearson | 2013 | World Journal of Translational Medicine2013，2，3： | 1 |
| 11 | Clinical and laboratoryfeatures of invasive community-onset methicillin-resistant Staphylococ-cus aureus infection : a prospective case-control study 显示文摘 | Wehrhahn MC Robinson JO Pearson JC | 2010 | Eur J ClinMicrobiol Infect Dis2010，29，8： | 1 |
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