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| 1 | Screeningfor amino acid substitutions in the Candida albicansErg11 protein of azole-susceptible and azole-resistantclinical isolates:new substitutions and a review ofthe literature显示文摘 | MORIO F LOGE C BESSE B | 2010 | Diagn Microbiol Infect Dis2010，66，4： | 1 |
| 2 | Increased VLDL-TAG turnover during and after acute moderateintensity exercise显示文摘 | Morio B Holmback U Gore D | | 0，，05： | 1 |
| 3 | Screening for amino acid substi- tutions in the Candida albicans Ergl 1 protein of azole-suscepti- ble and azole-resistant clinical isolates: new substitutions and a review of the literature 显示文摘 | Morio F Loge C Besse B | 2010 | Diagn Microbiol Infect Dis2010，66，4： | 1 |
| 4 | Screening for amino acid substi- tutions in the Candida albicans Ergl 1 protein of azole-suscepti- ble and azole-resistant clinical isolates: new substitutions and a review of the literature 显示文摘 | Morio F Loge C Besse B | 2010 | Diagn Microbiol Infect Dis2010，66，4： | 1 |
| 5 | Screening for amino acid substitutions in the Candida albicans Erg11 protein of azole-susceptible and azole-resistant clinical isolates:new substitutions and a review of the literature显示文摘 | Morio F Loge C Besse B | 2010 | Diagnostic Microbiology and Infectious Disease2010，66，4： | 1 |
| 6 | Mechanisms of body weight gain in patients with Parkinson's disease after subthalamic stimulation显示文摘 | Montaurier C Morio B Bannier S | 2007 | Brain2007，130，7： | 1 |
| 7 | Nutrition and protein energy homeostasis in elderly显示文摘 | Boirie Y Morio B Caumon E | 2014 | Mech Ageing Dev2014，1367，： | 1 |
| 8 | Mitochondrial dysfunction results from oxidative stress in the skeletal muscle of diet-induced insulin-resistant mice显示文摘 | Bonnard Charlotte Durand Annie Peyrol Simone Chanseaume Emilie Chauvin Marie-Agnes Morio Béatrice Vidal Hubert Rieusset Jennifer | 2008 | Journal of Clinical Investigation2008，，2： | 1 |
| 9 | Discussion of 'overweight and obese boys reduce food intake in response to a glucose drink but fail to increase intake in response to exercise of short du-ration'显示文摘 | Thivel D Duche P Morio B | 2012 | Appl Physiol Nutr Metab2012，37，5： | 1 |
| 10 | Pyrolysis kinetics of wheat and corn straw显示文摘 | MORIO L COLOMBA D B | 1998 | Journal of Analytica & Pyrolysis1998，，14： | 1 |
| 11 | Screening for amino acid substitutions in the Candida albicans Erg11 protein of azole-susceptible and azole-resistant clinical isolates:new substitutions and a review of the literature 显示文摘 | MORIO F LOGE C BESSE B | 2010 | Diagn Microbiol Infect Dis2010，66，： | 1 |
| 12 | Fat and protein redistribution with aging: metabolic considerations 显示文摘 | Beaufrere B Morio B | 2000 | Eur J Clin Nutr2000，54，： | 1 |
| 13 | Screening for amino acid substi- tutions in the Candida albicans Ergl 1 protein of azole - susceptible and azole - resistant clinical isolates : new substitutions and a re- view of the literature显示文摘 | Morio F Loge C Besse B | 2010 | Diagn Microbiol Infect Dis2010，66，4： | 1 |
| 14 | Maternal low protein diet induces persistent expression changes in metabolic genes in male rats显示文摘BACKGROUND Perinatal exposure to a poor nutritional environment predisposes the progeny to the development of metabolic disease at the adult age,both in experimental models and humans.Numerous adaptive responses to maternal protein restriction have been reported in metabolic tissues.However,the expression of glucose/fatty acid metabolism-related genes in adipose tissue and liver needs to be described.AIM To evaluate the metabolic impact of perinatal malnutrition,we determined malnutrition-associated gene expression alterations in liver and adipose tissue.METHODS In the present study,we evaluated the alterations in gene expression of glycolytic/Krebs cycle genes(Pyruvate dehydrogenase kinase 4 and citrate synthase),adipogenic and lipolytic genes and leptin in the adipose tissue of offspring rats at 30 d and 90 d of age exposed to maternal isocaloric low protein(LP)diet throughout gestation and lactation.We also evaluated,in the livers of the same animals,the same set of genes as well as the gene expression of the transcription factors peroxisome proliferator-activated receptor gamma coactivator 1,forkhead box protein O1 and hepatocyte nuclear factor 4 and of gluconeogenic genes.RESULTS In the adipose tissue,we observed a transitory(i.e.,at 30 d)downregulation of pyruvate dehydrogenase kinase 4,citrate synthase and carnitine palmitoyl transferase 1b gene expression.Such transcriptional changes did not persist in adult LP rats(90 d),but we observed a tendency towards a decreased gene expression of leptin(P=0.052).The liver featured some gene expression alterations comparable to the adipose tissue,such as pyruvate dehydrogenase kinase 4 downregulation at 30 d and displayed other tissue-specific changes,including citrate synthase and fatty acid synthase upregulation,but pyruvate kinase downregulation at 30 d in the LP group and carnitine palmitoyl transferase 1b downregulation at 90 d.These gene alterations,together with previously described changes in gene expression in skeletal muscle,may account for the metabolic adaptations in response to maternal LP diet and highlight the occurrence of persistent transcriptional defects in key metabolic genes that may contribute to the development of metabolic alterations during the adult life as a consequence of perinatal malnutrition.CONCLUSION We conclude that perinatal malnutrition relays long-lasting transcriptional alterations in metabolically active organs,i.e.,liver and adipose tissue. | Allan de Oliveira Lira JoséLuiz de Brito Alves Mariana Pinheiro Fernandes Diogo Vasconcelos David Filipe Santana João Henrique da Costa-Silva Béatrice Morio Carol Góis Leandro Luciano Pirola | 2020 | World Journal of Diabetes2020，11，5： | 0 |
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