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12篇 您的检索式：作者名="Helton ES"

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		题名	作者	年代	出处	被引量
	1	p53 modulation for DNA damage response显示文摘	Helton ES Chen X	2007	J Cell Biochem2007，100，4：	1
	2	Hormonal con- traception and HIV-1 infection: medroxyprogesterone acetate sup- presses innate and adaptive immune mechanisms 显示文摘	Huijbregts RP1 Helton ES Michel KG	2013	Endocri- nology2013，154，3：	1
	3	The proline-rich domain in p63 is necessary for the transcriptional and apoptosis-in- ducing activities of TAp63 显示文摘	Helton ES Zhang J Chen X	2008	Oncogene2008，，27：	1
	4	SURGISIS-assisted surgical site control in the delayed repair of a complex bile duct injury after laporoscopic cholecystectomy显示文摘	Ong ES Helton WS Jho D	2006	J Gastrointest Surg2006，10，2：	1
	5	SURGISIS-assisted surgical site control in the delayed repair of a complex bile duct injury after laparoscopic cholecystectomy显示文摘	Ong ES Helton WS Jho D	2006	J Gastrointest Surg2006，10，2：	1
	6	p53 modulation of the DNA damage response 显示文摘	Helton ES Chen X	2007	J Cell Biochem2007，100，4：	1
	7	r63 modulation of the DNA damage response显示文摘	Helton ES Chen X	2007	J Cell Biochem2007，100，4：	1
	8	Hormonal contraception and HIV-1 infection:medroxyprogesterone acetate suppresses innate and adap- tive immune mechanisms 显示文摘	Huijbregts RP Helton ES Michel KG	2013	Endocrinology2013，154，3：	1
	9	Immune suppression by neutrophils in HIV-1 infection:role of PD-L1/PD-1pathway显示文摘	Bowers NL Helton ES Huijbregts RP	2014	PLo S Pathog2014，10，10：	1
	10	SURGISIS-assisted surgical site control in the delayed repair of a complex bile duct injury after laparoscopic cholecystectomy显示文摘	Ong ES Helton WS Jho D	2006	J Gastrointest Surg2006，10，2：	1
	11	Thinking outside the channel: modeling nitrogen cycling in networked river ecosystems 显示文摘	Helton AM Poole GC Meyer JL Wollheim WM Peterson BJ Mulholland PJ Bernhardt ES Stanford JA Arango C Ashkenas LR	2010	Front Ecol Environ2010，9，：	1
	12	Self-reported participation restrictions in normal hearing individuals in thyroid ontogeny: Evidence of subclinical changes显示文摘Purpose: To verify evidence of subclinical alterations through self-reported participation restrictions in normal hearing individuals and congenital hypothyroidism patients.Methods: An analytical, quantitative, cross-sectional exploratory study with a 1:1 ratio, consisting of a convenience sample of 86 normal hearing individuals with(n = 42) and without(n = 44) congenital hypothyroidism diagnosis. All participants underwent tonal and speech audiometry, immitance and distortion product otoacoustic emissions. The researchers excluded people with hearing loss, genetic syndromes and metabolic diseases. The instrument used for evaluate of self-reported participation restrictions was the HHIE-adapted questionnaire, composed of 25 questions, 12 of which were social domain and 13 emotional domain. Student's ttest and chi-square test were used for statistical analysis at a significance level of 5%.Results: There was a significant(p < 0.001) self-reported participation restrictions in CH(61.9%), with a greater relevance for the social domain(p = 0.002). There was a greater frequency of mild/moderate(40.5%)and higher prevalence of association with clinical factors and adherence to treatment.Conclusion: The findings indicate that self-reported participation restrictions in normal hearing individuals with congenital hypothyroidism was more significant than in the non-exposed group, suggesting evidence of subclinical auditory abnormalities in this population.	Caio Le?nidas Oliveira de Andrade Gabriela Carvalho Machado Luan Paulo Franco Magalh?es Elen Pereira de Jesus Elaine Alves Matias Taíse Lima de Oliveira Cerqueira Hélida Braga de Oliveira Luciene da Cruz Fernandes Helton Estrela Ramos Crésio de Arag?o Dantas Alves	2019	Journal of Otology2019，14，1：	0

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