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23篇 您的检索式:作者名="Engels IH"
    题名 作者 年代 出处 被引量
1Apoptosis resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of caspase-3 and a caffeine-inhibitable event显示文摘Essmann F Engels IH Totzke G 2004Cancer Res2004,64,19:1
2Anticancer drugs induce caspase 8/FLICE activation and apoptosis in the absence of CD95 receptor/ligand interaction 显示文摘Wesselborg S Engels IH Rossmann E 1999Blood1999,93,:1
3Hypertension due to renal compression resulting from subcapsular hematoma显示文摘ENGEL WJ PAGE IH 1955J Urol1955,73,5:1
4Apoptosis resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of caspase-3 and a caffeine-inhibitable event显示文摘Essmann F Engels IH Totzke G 2004Cancer Res2004,64,19:1
5Human mature red blood cells express caspase-3 and caspase-8, but are devoid of mitochondrial regulators of apoptosis显示文摘Berg CP Engels IH Rothbart A 2001Cell Death Differ2001,8,12:1
6Mistletoe leetin activates caspese-8/FLICE independently of death receptor signaling and enhances anticancer drug-induced apoptesis显示文摘Bantel H Engels IH Voelter W 1999Cancer Res1999,59,9:1
7Caspase-lO Sensitizes Breast Carcinoma Cells to TRAIL-Induced but Not Tumor Necrosis Factor-Induced Apoptosis in a Caspase-3-Dependent Manner显示文摘Engels IH Totzke G Fischer U 2005Mol Cell Biol2005,25,7:1
8Synthetic lethal targeting of MYC by activation of the DR5 death receptor pathway显示文摘Wang Y Engels IH Knee DA 2004Cancer Cell2004,5,:1
9Apoptosis Resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of Caspase-3 and a caffeine-inhibitable evevnt显示文摘Essmann F Engels IH Totzke G 2004Cancer Res2004,64,19:1
10Expression of calcium-binding proteins MRP8 and MRP14 in inflammatory muscle diseases显示文摘Seeliger S Vogl T Engels IH 2003Am J Pathol2003,163,3:1
11Apoptosis Resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of Caspase-3 and a caffeine-inhibitable evevnt 显示文摘Essmann F Engels IH Totzke G 2004Cancer Res2004,64,19:1
12Hypertension due to renal compression resulting from subeapsular hematoma 显示文摘Engel WJ Page IH 1955Urol1955,73,5:1
13Caspase-2 can function upstream of bid cleavage in the TRAIL apoptosis pathway显示文摘Wagner KW Engels IH Deveraux QL 2004J Biol Chem2004,279,35:1
14caspase-10 sensitizes breast carcinoma cells to TRAIL-induced but not tumor necrosis factor-induced apoptosis in a easpase -3-dependent manner显示文摘Engels IH Totzke G Fischer U et el 2005Mol Cell Bio12005,25,7:1
15Human mature red blood cells express caspase-3 and easpase-8, but are devoid of mitochondrialregulators of apoptosis显示文摘Berg CP Engels IH Rothbart A 2000Cell Death Differ2000,8,12:1
16Apoptosis resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of caspase-3 and a caffeine-inhibitable event显示文摘Essmann F Engels IH Totzke G 2004CancerRes2004,64,19:1
17Apoptosis resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of easpase-3 and a caffeine-inhibitable event显示文摘 Engels IH Totzke G 2004Cancer Res2004,64,:1
18Caspase-2 can function upstream of bid cleavage in the TRAIL apoptosis pathway 显示文摘Wagner KW Engels IH Deveraux QL 2004J Biol Chem2004,279,35:1
19Expression of calcium- binding proteins MRP8 and MRP14 in inflammatory muscle diseases显示文摘Seeliger S Vogl T Engels IH 2003Am J Pathol2003,163,3:1
20Calgranulin B(S100A9)levels in bronchoalveolar lavage fluid of patients with interstitial lung diseases显示文摘Seeliger S Vogl T Engels IH 0,,05:1
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