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12篇 您的检索式：作者名="Empson M"

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		题名	作者	年代	出处	被引量
	1	Recurrent pregnancy loss with antiphospholipid antibody:a systematic review of therapeutic trials显示文摘	Empson M Lassere M Craig JC	2002	Obstet Gynecol2002，99，1：	1
	2	Atopy,anergic status,and cytokine expression in HIV-infected subjects显示文摘	Empson M Bishop GA Nightingale B	1999	J Allergy Clin Immunol1999，103，1：	1
	3	Recurrent pregnancy loss with antiphospholipid antibody a systematic review of therapeutic trials显示文摘	Empson M Lassere M Craig JC	2002	Obstet Gynecol2002，99，1：	1
	4	Risk factors for colonization with vancomyein-resistant enterococei in a Melbourne hospital 显示文摘	MacIntyre CR Empson M Boardman C	2001	Infect Control Hosp Epidemiol2001，22，10：	1
	5	Prevalence of thyroiddisease in an older Australian population 显示文摘	Empson M Flood V Ma G	2007	Intern Med J2007，37，7：	1
	6	Relative toxieity and metaholic effects of c, holecalciferol and 25-hy- droxycholecalciferol in chicks显示文摘	Morrissey R L Colin R M Empson R N Jr	1977	Journal of Nutrition1977，107，：	1
	7	Too much of a good thing, is it bad? Adrenaline on trial 显示文摘	Sood JD Empson M Fitzharris P	2007	N Z Med J2007，120，1252：	1
	8	Prevention of recurrent miscarriage for women with antiphospholi~id antibody or lupus anticoagulant显示文摘	Empson M Lassere M Craig J	2005	Cochrane Database Syst Rev2005，，00：	1
	9	Recurrent pregnancy loss with antiphospholipid antibody:a systematic review of therapeutic trials显示文摘	Empson M Lassere M Craig JC Scott JR	2002	Obstet Gynecol2002，99，1：	1
	10	Prevention of recurrent rnlsearriage for women with antiphospholipid antibody or lupus anticoagulant显示文摘	EMPSON M LASSERE M CRAIG J	2005	Coehrane Database Syst Rev2005，4，00：	1
	11	Adenosine acting via A1 receptors, controls the transition to status epilepticus-like behavior in an in vitro model of epilepsy显示文摘	Acsar E Empson R M	2004	Neuropharmaco[ogy2004，47，3：	1
	12	Contribution of plasma membrane Ca^(2+) ATPase to cerebellar synapse function显示文摘The cerebellum expresses one of the highest levels of the plasma membrane Ca2+ATPase,isoform 2 in the mammalian brain.This highly efficient plasma membrane calcium transporter protein is enriched within the main output neurons of the cerebellar cortex;i.e. the Purkinje neurons(PNs) .Here we review recent evidence,including electrophysiological and calcium imaging approaches using the plasma membrane calcium ATPase 2(PMCA2) knockout mouse,to show that PMCA2 is critical for the physiological control of calcium at cerebellar synapses and cerebellar dependent behaviour.These studies have also revealed that deletionof PMCA2 throughout cerebellar development in the PMCA2 knockout mouse leads to permanent signalling and morphological alterations in the PN dendrites. Whilst these findings highlight the importance of PMCA2 during cerebellar synapse function and development,they also reveal some limitations in the use of the PMCA2 knockout mouse and the need for additional experimental approaches including cell-specific and reversible manipulation of PMCAs.	Helena Huang Raghavendra Y Nagaraja Molly L Garside Walther Akemann Thomas Knpfel Ruth M Empson	2010	World Journal of Biological Chemistry2010，1，5：	0

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