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		题名	作者	年代	出处	被引量
	1	Mechanisms of neuronal damage in multiple sclerosis and its animal models: role of Calcium pumps and exchangers显示文摘	Kumellas MP Donahue KC Elkabes S	2007	Biochem Soc Trans2007，35，5：	1
	2	Gene expession in activated brain microglia: Identification of a proteinase inhibitor that increase microglial cell number显示文摘	Smita TV Elkabes S Schick C	1998	Mol Brain Res1998，56，：	1
	3	Role of plasma membrane calcium ATPase 2 in spinal cord pathology显示文摘A number of studies have indicated that plasma membrane calcium ATPases(PMCAs) are expressed in the brain and spinal cord and could play important roles not only in the maintenance of cellular calcium homeostasis but also in the survival and function of central nervous system cells under pathological conditions.The different regional and cellular distributions of the various PMCA isoforms and splice variants in the nervous system and the diverse phenotypes of PMCA knockout mice support the notion that each isoform might play a distinct role. Especially in the spinal cord,the survival of neurons and,in particular,motor neurons could be dependent on PMCA2.This is indicated by the knockdown of PMCA2 in pure spinal cord neuronal cultures that leads to cell death via a decrease in collapsing response mediator protein 1 levels.Moreover,the progressive decline in the number of motor neurons in PMCA2-null mice andheterozygous mice further supports this notion.Therefore,the reported reduction in PMCA2 mRNA and protein levels in the inflamed spinal cord of mice affected by experimental autoimmune encephalomyelitis(EAE) ,an animal model of multiple sclerosis,and after spinal cord contusion injury,suggests that changes in PMCA2 expression could be a cause of neuronal pathology and death during inflammation and injury.Glutamate excitotoxicity mediated via kainate receptors has been implicated in the neuropathology of both EAE and spinal cord injury,and has been identified as a trigger that reduces PMCA2 levels in pure spinal cord neuronal cultures through degradation of the pump by calpain without affecting PMCA2 transcript levels.It remains to be determined which other stimuli modulate PMCA2 mRNA expression in the aforementioned pathological conditions of the spinal cord.	Amanda Kathleen Fakira Stella Elkabes	2010	World Journal of Biological Chemistry2010，1，5：	1
	4	Gene expession in activated brain microglia:identification of a p roteinase inhibitor that increase micro- glial cell number显示文摘	mita TV Elkabes S Schick C	1998	Mol Brain Res J1998，56，：	1
	5	Brain microglia/ macro phages express neurotrophins that selectively regulate microglial pro-liferation and function显示文摘	Elkabes S DiCicco-Bloom EM Black IB	1996	J Neurosci1996，，16：	1
	6	Brain microglia/macrophages express neurotrophins that selectively regulate microglial proliferation and function显示文摘	Elkabes S DiCicco-Bloom EM Black IB		0，，：	1
	7	Evidence for the presence of N - acetylaspartylglutamate in cultured oligodendrocytes and LPS activated microglia 显示文摘	Passani L Elkabes S Coyle JT	1998	Brain Res1998，794，1：	1
	8	Brain microglia/macrophages express neu- rotrophins that selectively regulate microglial proliferation and function 显示文摘	Elkabes S DiCicco-Bloom EM Black lB	1996	J Neurosci1996，16，：	1
	9	Prevalence of hepatitis C virus antibodies among different populations of relative and attributable risk显示文摘	Daw MA Elkaber MA Drah AM	2002	Saudi Med J2002，23，11：	1
	10	Gene expression in activated brain microglia: identification of a proteinase inhibitor that increase microglial cell number 显示文摘	Elkabes S Schick C	1998	Mol Brain Res1998，56，：	1
	11	CagA positivity and its association with gastroduodenal disease in Turkish children undergoing endoscopic investigation显示文摘	Semra S?kücü Ay?e Tülin ?zden ?zlem Durmaz Süo?lu Berna Elkabes Fikri Demir U?ur ?evikba? Selim G?k?e Günay Saner	2006	Journal of Gastroenterology2006，，6：	1

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