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34篇 您的检索式:作者名="Cremolini C"
    题名 作者 年代 出处 被引量
1FOLFOXIRI plus bevacizumab versus FOLFIRI plus bevacizumab as first-line treatment of patients with metastatic colorectal cancer:updated overall survival and molecular subgroup analyses of the open-label,phase 3 TRIBE study 显示文摘Cremolini C Loupakis F Antoniotti C 2015Lancet Oncol2015,16,13:1
2Prospective validation of candidate SNPs of VEGF/VEGFR pathway in metastatic colorectal cancer patients treated with first-line FOLFIRI plus bevacizumab显示文摘Loupakis F Cremolini C Yang D 2013PLo S One2013,8,66:1
3Early tumour shrinkage as a prognostic factor and surrogate end - point in colorectal cancer: A systematic review and pooled - analysis显示文摘Petrelli F Pietrantonio F Cremolini C 2015Eur J Cancer2015,51,7:1
4KRAS codon61,146 and BRAF mutations predict resistance to cetu-ximab plus irinotecan in KRAS codon 12 and 13 wild-typemetastatic colorectal cancer显示文摘Loupakis F Ruzzo A Cremolini C 2009Br J Cancer2009,101,4:1
5Clinical impact of anti - epidermal growth factor receptor monoclonal an- tibodies in first - line treatment of metastatic colorectal cancer显示文摘LOUPAKIS F CREMOLINI C SALVATORE L 2012Cancer2012,118,6:1
6FOLFOXIRI/bevacizum- ab (bey) versus FOLFIRI/bev as first-line treatment in unresect- able metastatic colorectal cancer (mCRC) patients (pts) : Results of the phase Ⅲ TRIBE trim by GONO group 显示文摘Faocone A Cremolini C Masi G 2013J Clin Oncol2013,,:1
7FOLFOXIRI/bevacizumab(bev) versus FOLFIRI/bev as first-line treatment in unresectable metastatic colorectal cancer (mCRC) patients (pts):Results of the phase Ⅲ TRIBE trial by GONO group显示文摘Falcone A Cremolini C Gianluca M 2013J Clin Oncol2013,31,15:1
8Initial therapy with FOLFOXIRI and bevacizumab for metastatic colorectal cancer 显示文摘Loupakis F Cremolini C Masi G 2014N EnglJ Med2014,371,17:1
9Predictors of benefit in colorectal cancer treated with cetuximab:are we getting 'Lost in TranslationAL'显示文摘Cremolini C Loupakis F Ruzzo A 2010J Clin Oncol2010,28,11:1
10KRAS codon 61,146 and BRAF mutations predict resistance to cetuximab plus irinotecan in KRAS codon 12 and 13 wild - type metastatic colorectal cancer 显示文摘Loupakis F Ruzzo A Cremolini C 2009Br J Cancer2009,101,:1
11FOLFOXIRI plus bevacizumab as first-line treatment in BRAF mutant metastatic colorectal cancer显示文摘Loupakis F Cremolini C Salvatore L 2014Eur J Cancer2014,50,:1
12First-line anti- EGFR monoclonal antibodies in panRAS wild-type metastatic colorectal cancer: A systematic review and recta-analysis 显示文摘Pietrantonio F Cremolini C Petrelli F 2015Crit Rev Oncol Hematol2015,96,1:1
13Initial therapy with FOLFOXIRI and bevacizumab for metastatic eolorectal cancer显示文摘LOUPAKIS F CREMOLINI C MASI G 2014N Engl J Med2014,,:1
14Initial therapy with FOLFOXIRI and bevacizumab for metastatic colorectal cancer显示文摘LOUPAKIS F CREMOLINI C MASI G 2014N Engl J Med2014,371,17:1
15Initial therapy with FOLFOXIRI and bevacizumab for metastatic colorectal cancer显示文摘Loupakis F Cremolini C Masi G 2014N Engl J Med2014,371,17:1
16FOLFOXIRI/ bevacizumab(bev)versus FOLFIRI/bev as first-line treatment in unresectable metastatic colorectal cancer(mCRC)patients(pts):Results of the phase Ⅲ TRIBE trial by GONO group MD显示文摘FALCONE A CREMOLINI C MASI G 2013J Clin Oncol2013,31,15:1
17Initial therapy with FOLFOXIRI and bevacizumab for metastatic colorectal cancer 显示文摘Loupakis F Cremolini C Masi G 2014N Engl J Med2014,371,17:1
18KRAS codon 61, 146and BRAF mutations predict resistance to cetuximab plusirinotecan in KRAS codon 12 and 13 wild-type metastaticcolorectal cancer显示文摘Loupakis F Ruzzo A Cremolini C 2009Br J Cancer2009,101,4:1
19FOLFOXIRI plus bevacizumab versus FOLFIRI plus bevacizumab as first-line treatment of patients with metastatic colorectal cancer:updated overall survival and molecular subgroup analyses of the open-label,phase3 TRIBE study显示文摘Cremolini C Loupakis F Antoniotti C 2015Lancet Oncol2015,16,13:1
20Subgroup analyses in RAS mutant,BRAF mutant and all-wt mC RC pts treated with FOLFOXIRI plus bevacizumab(bev)or FOLFIRI plus bev in the TRIBE study显示文摘Loupakis F Cremolini C Lonardi S 2014J Clin Oncol2014,32,:1
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