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44篇 您的检索式:作者名="Brasacchio"
    题名 作者 年代 出处 被引量
1Advanced glycation end-products induce vascular dysfunction via resistance to nitric oxide and suppression of endothelial nitric oxide synthase显示文摘Aino Soro-Paavonen Wei-Zeng Zhang Kylie Venardos Melinda T Coughlan Emma Harris David CK Tong Daniella Brasacchio Karri Paavonen Jaye Chin-Dusting Mark E Cooper David Kaye Merlin C Thomas Josephine M Forbes 2010Journal of Hypertension2010,,4:1
2Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
3Hyperglycemia induces a dynamic cooperativity of histone methylase and demethylase enzymes associated with gene - activating epigenetic marks that coexist on the lysine tail显示文摘Brasacchio D Okabe J Tikellis C 2009Diabetes2009,58,5:1
4Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,:1
5Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘EI-Osta A Brasacchio D Yao D 2008Journal of Experimental Medicine2008,205,10:1
6Hyperglycemia induces a dy- namic cooperativity of histone methylase and demethylase enzymes associated with gene-aetivating epigenetic marks that coexist on the lysine tail 显示文摘BRASACCHIO D OKABE J TIKELLIS C BALCERCZYK A GEORGE P BAKER EK 2009Diabetes2009,58,5:1
7Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
8Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao Dj 2008J Exp Med2008,205,10:1
9Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia 显示文摘EL-OSTA A BRASACCHIO D YAO D C 2008The Journal of Experimental Med- icine2008,205,10:1
10Transient high glucose causes peristent epigenetic changes and altered gene expression during subsequent normoglycemia 显示文摘E1-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
11Hyperglycemia in duces a dynamic cooperativity of histone methylase and demethylase enzymes associated with gene activating epigenetic marks that coexist on the lysine tail显示文摘Brasacchio D Okabe J Tikellis C 2009Diabetes2009,58,5:1
12Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘EI-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
13Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
14Transient high glucose cau- ses persistent epigenetic changes and altered gene expression during sub -sequent normoglyeemia 显示文摘El - Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
15Transient high glucose causes persistent epigenetic changes mad altered gene expression during sub- sequent normoglycemia 显示文摘EI-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,:1
16Transient high glucose cauges persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘EI-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
17Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
18Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia 显示文摘el OSTA A BRASACCHIO D YAO D 2008J Exp Med2008,205,10:1
19Transient high glucosecauses persistent epigenetic changes and altered gene expressionduring subsequent normoglycemia显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
20Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia 显示文摘El-Osta A Brasacchio D Yao D 2008J Exp Med2008,205,10:1
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