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10篇 您的检索式:作者名="Barry GE"
    题名 作者 年代 出处 被引量
1In vitro differentiation potential of human embryonic versus adult stem cells显示文摘Rooney GE Nistor GI Barry FB 0,,03:1
2In vitro differentiation potential of human embryonic versus adult stem cells显示文摘Rooney GE Nistor GI Barry FB 2010Regen Med2010,5,3:1
3Age-related differences in rapid muscle activation after rate of force development training of the elbow flexors 显示文摘Barry BK Warman GE Carson RG 2005Exp Brain Res2005,162,1:1
4Potential of rat bone marrow-derived mesenchymal stem cells as vehicles for delivery of neurotrophins to the Parkinsonian rat brain 显示文摘Moloney TC Rooney GE Barry FP 2010Brain Res2010,1359,:1
5Leydig cells:from stem cells to aging显示文摘Haolin Chena Ren-Shan Ge Barry RZ 2009Mol Cell Endocrinol2009,306,12:1
6Potential of rat bone marrow-derived mesenchymal stem cells as vehicles for delivery of neurotrophins to the Parkinsonian rat brain显示文摘Moloney TC Rooney GE Barry FP 2010Brain Res2010,1359,:1
7In vitro differentiationpotential of human embryonic versus adult stem cells显示文摘Rooney GE Nistor GI Barry FB 0,,03:1
8New solid - phase enzyme immunoassay of neuron - specific enolase in serum : effect of storage, temprature, lipemia, icterus and hemolysis 显示文摘Mercer DW Virji MA Barry GE 1990Clin Chem1990,36,8:1
9Age-related differences in rapid muscle activation after rate of force development training of the elbow flexors显示文摘Barry BK Warman GE Carson RG 2005Exp Brain Res2005,162,1:1
10The collagen receptor,discoidin domain receptor 2,functions in Gli1-positive skeletal progenitors and chondrocytes to control bone development显示文摘Discoidin Domain Receptor 2(DDR2)is a collagen-activated receptor kinase that,together with integrins,is required for cells to respond to the extracellular matrix.Ddr2 loss-of-function mutations in humans and mice cause severe defects in skeletal growth and development.However,the cellular functions of Ddr2 in bone are not understood.Expression and lineage analysis showed selective expression of Ddr2 at early stages of bone formation in the resting zone and proliferating chondrocytes and periosteum.Consistent with these findings,Ddr2^(+)cells could differentiate into hypertrophic chondrocytes,osteoblasts,and osteocytes and showed a high degree of colocalization with the skeletal progenitor marker,Gli1.A conditional deletion approach showed a requirement for Ddr2 in Gli1-positive skeletal progenitors and chondrocytes but not mature osteoblasts.Furthermore,Ddr2 knockout in limb bud chondroprogenitors or purified marrow-derived skeletal progenitors inhibited chondrogenic or osteogenic differentiation,respectively.This work establishes a cell-autonomous function for Ddr2 in skeletal progenitors and cartilage and emphasizes the critical role of this collagen receptor in bone development.Fatma F.Mohamed Chunxi Ge Randy T.Cowling Daniel Lucas Shawn A.Hallett Noriaki Ono Abdul-Aziz Binrayes Barry Greenberg Renny T.Franceschi 2022Bone Research2022,10,1:0
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