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| 1 | Free fatty acids sensi- tise hepatocytes to TRAIL mediated cytotoxicity 显示文摘 | MALHI H BARREYRO FJ ISOMOTO H | 2007 | Gut2007，56，8： | 1 |
| 2 | Stem and progenitor cells in myelodysplastic syndromes show aberrant stage-specific expansion and harbor genetic and epigenetic alterations显示文摘 | Britta Will Li Zhou Thomas O. Vogler Susanna Ben-Neriah Carolina Schinke Roni Tamari Yiting Yu Tushar D. Bhagat Sanchari Bhattacharyya Laura Barreyro Christoph Heuck Yonkai Mo Samir Parekh Christine McMahon Andrea Pellagatti Jacqueline Boultwood Cristina | 2012 | Blood2012，，10： | 1 |
| 3 | Free fatty acids sensitise hepatocytes to TRAIL mediated cytotoxieity显示文摘 | Malhi H Barreyro F J Isomoto H | 2007 | Gut2007，56，8： | 1 |
| 4 | TRAIL mediates liver injury by the innate immune system in the bile duct-ligated mouse显示文摘 | Kahraman A Barreyro F J Bronk S F | 2008 | Hepatology2008，47，4： | 1 |
| 5 | Overexpression of IL-1 receptor accessory protein in stem and progenitor cells and outcome correlation in AML and MDS 显示文摘 | Laura Barreyro Britta Will Boris Bartholdy | 2012 | Blood2012，120，： | 1 |
| 6 | Transcrip- tional regulation of Bim by FoxO3A mediates hepatocyte lipoapopto- sis 显示文摘 | BARREYRO F J KOBAYASHI S BRONK S F | 2007 | Biol Chem2007，282，27： | 1 |
| 7 | Free fatty acids sensitise hepatocytes to TRAIL mediated cytotoxieity 显示文摘 | Malhi H Barreyro FJ Isomoto H | 2007 | Gut2007，56，8： | 1 |
| 8 | Transcriptional regulation of Bim by FoxO3A mediates hepatocyte lipoapoptosis显示文摘 | Kobayashi S Bronk SF | 2007 | J Biol Chem2007，282，27： | 1 |
| 9 | Estimation of endogenous protein and amino acid ileal losses in weaned piglets by regression analysis using diets with graded levels of casein显示文摘Background: Many studies have investigated endogenous loss of proteins and amino acids(AAs) at the ileal level in growing pigs. However, only a few studies have researched this subject in piglets. Knowledge regarding AA ileal digestibility in piglets would be helpful during the formulation of diets for weaning piglets, rather than just using coefficients obtained in growing pigs. Therefore, in this study, we sought to estimate endogenous protein and AA ileal losses in piglets. Furthermore, apparent and true ileal digestibility(AID and TID) of protein and AAs from casein were measured.Results: The average flow of protein was 20.8 g/kg of dry matter intake(DMI). Basal protein loss, as estimated by regression, was 16.9 g/kg DMI. Glutamic acid, arginine, and aspartic acid(2.2, 1.4, and 1.2 g/kg DMI, respectively)were the AAs for which greater losses were seen. The AID of protein and AAs increased as the protein level in the diet increased. A higher increment in AID was observed between diets with 80 and160 g CP/kg of feed; this finding was mainly attributable to increases in glycine and arginine(46.1% and 18%, respectively). The TID of protein was97.8, and the TID of AAs varied from 93.9 for histidine to 100.2 for phenylalanine.Conclusions: The basal endogenous protein loss in piglets was 16.9 g/kg DMI. Endogenous protein was rich in glutamic acid, aspartic acid, and arginine, which represented 32.7% of endogenous protein loss in weaning piglets.The TID of casein was high and varied from 93.0 for histidine to 100.2 for phenylalanine. | Tércia Cesária Reis de Souza Araceli Aguilera Barreyro Gerardo Mariscal-Landín | 2014 | Journal of Animal Science and Biotechnology2014，5，1： | 0 |
| 10 | Hepatocellular carcinoma in patients with metabolic dysfunctionassociated fatty liver disease: Can we stratify at-risk populations?显示文摘Metabolic dysfunction-associated fatty liver disease(MAFLD)is a new nomenclature recently proposed by a panel of international experts so that the entity is defined based on positive criteria and linked to pathogenesis,replacing the traditional non-alcoholic fatty liver disease(NAFLD),a definition based on exclusion criteria.NAFLD/MAFLD is currently the most common form of chronic liver disease worldwide and is a growing risk factor for development of hepatocellular carcinoma(HCC).It is estimated than 25%of the global population have NAFLD and is projected to increase in the next years.Major Scientific Societies agree that surveillance for HCC should be indicated in patients with NAFLD/MAFLD and cirrhosis but differ in non-cirrhotic patients(including those with advanced fibrosis).Several studies have shown that the annual incidence rate of HCC in NAFLD-cirrhosis is greater than 1%,thus surveillance for HCC is costeffective.Risk factors that increase HCC incidence in these patients are male gender,older age,presence of diabetes and any degree of alcohol consumption.In non-cirrhotic patients,the incidence of HCC is much lower and variable,being a great challenge to stratify the risk of HCC in this group.Furthermore,large epidemiological studies based on the general population have shown that diabetes and obesity significantly increase risk of HCC.Some genetic variants may also play a role modifying the HCC occurrence among patients with NAFLD.The purpose of this review is to discuss the epidemiology,clinical and genetic risk factors that may influence the risk of HCC in NAFLD/MAFLD patients and propose screening strategy to translate into better patient care. | Eduardo Fassio Fernando J Barreyro M Soledad Pérez Diana Dávila Graciela Landeira Gisela Gualano Gabriela Ruffillo | 2022 | World Journal of Hepatology2022，14，2： | 0 |
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